
Assistant Professor
Department of Psychology
(360) 546-9748
Education: Ph.D.,Oregon Health & Science University,
Pharmacology and Neuroscience
Courses Offered:
Neuro 301/Bio 492 - Introduction to Neuroscience: Exploring the
Brain Ð The brain controls all of
our lives; what we sense, how we move, and what we think. The study of the brain (or
neuroscience) is interdisciplinary in that it encompasses diverse scientific
disciplines such as medicine, biology, psychology, molecular biology, physics,
chemistry and mathematics.
Neuroscience is the fastest growing discipline in experimental biology
throughout the world. This course
surveys the many aspects of neuroscience and lays down the foundation for
understanding the nervous system control of human behaviors.
Bio 407 - Biology of Women - This
course will examine biological sex differentiation and reproductive anatomy. It
will cover women's reproductive life from menarche to menopause, including such
topics as birth control, infertility, and pregnancy. Women's health issues such
as sexually transmitted diseases, breast cancer, eating disorders,
osteoporosis, and heart disease will also be covered. Biol/WSt 407 fulfills
three hours of biological science (B) or three hours of Tier III capstone (T)
for General Education Requirements (GER) for graduation.
Research Areas:
Neural
Mechanisms of Morphine Tolerance and Dependence
Chronic
morphine administration induces adaptations in neurons resulting in opioid
tolerance and dependence. The
periaqueductal gray area (PAG) neurons are an integral part of the descending
pathway that modulates pain impulses in the spinal cord and these neurons are
implicated in the expression of many behavioral signs of opioid
withdrawal. However, the cellular
mechanisms underlying the changes observed after chronic opioid administration
are not understood. My laboratory
uses whole-cell patch-clamp recordings from brain-slices of the PAG to study
the cellular mechanisms of opiate tolerance and dependence. We have previously found that chronic
morphine treatment changes the efficacy of coupling of µ-opioid receptors to
their effectors resulting in an increased release of GABA from PAG
neurons. We are interested in
further exploring these changes within the context of the involvement of PAG
neurons in the descending pain pathway and their resulting effects on pain
modulation. We are collaborating
with the laboratory of Dr. Michael Morgan so that we might correlate cellular
changes in the PAG with behavioral measures of pain and tolerance to morphine.
Regulation of Synaptic Transmission in the Midbrain by the
Dopamine Transporter
The
laboratory is also interested in the cellular mechanisms associated with other
drugs of abuse such as psychostimulants (cocaine and amphetamines). The main cellular target for cocaine
and amphetamine are the monoamine neurotransmitter transporters. These transporters are also the targets
for therapeutic drugs for disorders such as depression and attention deficit
disorder. Although the
transporters are primarily known for regulating the extracellular
concentrations of neurotransmitters through reuptake of released neurotransmitters,
they have significant electrical activities as well. Our recent studies of the electrical properties of the
monoamine transporters have shown that these transporters have a potential role
in modulating excitability of midbrain neurons (see recent publications). Future studies are planned to
determine the relevance of transporter-associated currents in the regulation of
synaptic transmission in monoaminergic neurons associated with reward pathways
within the midbrain.
Publications
16. Ingram, S.L., Prasad, B.M.
and Amara, S.G. (2002) Dopamine transporter-mediated conductances increase
excitability of midbrain dopamine neurons. Nature Neuroscience 5:971-8.
17. Connor, M.,
Bagley, E.E., Mitchell, V.A., Ingram, S.L., Christie, M.J., Humphrey, P.A., and Vaughan, C.W. (2004)
Cellular actions of somatostatin on rat periaqueductal grey neurons in vitro. British
Journal of Pharmacology doi:10.1038/sj.bjp.0705894
18. Morgan, M.M.,
Tierney, B.W. and Ingram,
S.L.
(2005). Intermittent dosing prolongs tolerance to the antinociceptive effect of
morphine microinjection into the periaqueductal gray. Brain Research, 1059:173-178.
19. Morgan, M.M.,
Fossum, E.N., Levine, C.S., & Ingram, S.L. (2006). Antinociceptive tolerance revealed by
cumulative intracranial microinjections of morphine into the periaqueductal
gray in the rat. Pharmacology, Biochemistry & Behavior. 85(1):214-219
20. Heinricher, M.M. and Ingram, S.L. (2007).
The brainstem and nociceptive modulation. Handbook of the Senses, Academic Press: Elsevier (London), in press.
21. Saugstad, J.A. & Ingram, S.L. (2007). Group I Metabotropic Glutamate Receptors.
Humana Press, Inc., in press.
22. Ingram, S.L., Fossum, E.N., & Morgan, M.M. (2007). Behavioral and
electrophysiological evidence for opioid tolerance in adolescent rats. Neuropsychopharmacology. Mar;32(3):600-6. Epub 2006 Jun 28.
23. Meyer, P.J., Fossum, E.N., Ingram, S.L. & Morgan, M.M. (2007). Analgesic tolerance to microinjection of the µ-opioid
agonist DAMGO into the ventrolateral periaqueductal gray. Neuropharmacology.
52(8):1580-5. Epub 2007 Mar 12.
24. Ingram, S.L., Macey, T.A., Fossum, E.N., & Morgan, M.M. (2007). Increased potency of opioid agonists is associated with tolerance to repeated morphine administration. Neuropsychopharmacology advance online publication 28 November 2007; doi: 10.1038/sj.npp.1301634.
25. Fossum, E.N., Lisowski, M., Macey, T.A., Ingram, S.L., & Morgan, M.M. (2008). Repeated administration of DMSO reduces the antinociceptive potency of morphine microinjected into the PAG. Brain Research, In press.